Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Conversely, our power to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK signifies elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by increased pFAK localization in five-HT synapses. https://pro3355544.blogdosaga.com/32197358/fascination-about-pro33